Canonical Question

Blood – Consequences

V5 N4.iii Historical V4 Q4.ii 5 appearances
Variant family: at least one exam appearance has different scope. The appearance record controls whether this master answer is safe to display on that Past Paper.

Master answer

Adverse Consequences of Blood Transfusion

1. Acute (<24 hours)

1a) Immune-mediated
ProblemMethods to Minimize
Allergic reaction to plasma proteinsmild (urticarial) or severe (anaphylaxis)Pre-treatment in high risk patients
Acute Haemolytic transfusion reactionincompatibility of donor and recipient blood leads to widespread haemolysis and circulatory collapseGroup and screening
Febrile non-haemolytic transfusion reaction (FNHTR)due to stored cytokines and/or the presence of recipient alloantibodiesCareful monitoring and early cessation of transfusion  
Transfusion related acute lung injury (TRALI)noncardiogenic pulmonary oedema caused by HLA antibodies in donor plasma directed against recipient leukocytes or bioactive lipids which accumulate during storageCareful monitoring and early cessation of transfusion

1b) Non-immune mediated
Problem Methods to Minimize
Sepsisbacterial infections are most common with platelets as they are stored at room temperatureSafe and sterile methods of storage
Transfusion Related Circulatory overload (TACO)fluid overload usually due to rapid or massive transfusionCareful monitoring and early cessation of transfusion
Non-immune mediated haemolysisCareful monitoring and early cessation of transfusion
HypothermiaCareful monitoring and early cessation of transfusion
Dilutional coagulopathy  Careful monitoring and early cessation of transfusion

2. Delayed (>24 hours)

2a) Immune-mediated
Problem Methods to Minimize
Delayed haemolytic transfusion reactionProduction of anti-donor antibodies post-transfusion
May be associated with transfused malaria
Careful monitoring and early cessation of transfusion
Transfusion-related immunomodulation (TRIM)transient immunosuppression in blood recipients which may be due to release of cytokines from donor lymphocytesCareful monitoring and early cessation of transfusion
Alloimmunisationdevelopment of antibodies during exposure to blood products, resulting in an amplified reaction on subsequent exposure

Can cause post transfusion thrombocytopenia and purpura
Re-screening every 48-72 hours
Transfusion Associated Graft vs. Host DiseaseProfound bone marrow aplasia
>90% mortality
Viable donor T cells implant and attack recipient tissues
Careful monitoring.
Re-screening

2b) Non-immune mediated
Problem Methods to Minimize
Iron overloadmost common in chronically transfused patients Monitoring of Iron levels
Transfusion-related infectionViral – the risk of HIV, HTLV 1&2 and HCV is <1/1 million. The risk of contracting HBV is slightly higher at 1/500,000.

Other – malaria, vCJD, Dengue Fever, West Nile Virus
Pre-screening of donors, screening of collected blood as per local guidelines (different for different areas)

3. Storage Lesions

A storage lesion refers to the changes that occur to a sample of blood during storage. (Note: Australian Red cross anti-coagulates Whole blood with CPD, but washes and stores Red Cells in SAGM).

Problem Methods to Minimize
Physical changesReduction in the viability of RBCs due to shape changes and reduced deformability

Formation of microaggregates
Careful monitoring and usage of products within a specified period    
Hyperkalaemiaplasma K can be >20 at 28 days in stored blood due to inactivation of the red cell Na/K ATPase pump.Careful monitoring and usage of products within a specified period
High citrate loadcan lead to hypocalcaemia and alkalosis (less or nil in SAGM)Use of SAGM for storage
Reduction in 2,3-BPGcauses left shift of the oxygen/haemoglobin dissociation curve (less in CPDA1)Use of CPDA1    
Renal impairmentDue to Increase in free haemoglobin from cell lysisRoutine monitoring of renal function and careful monitoring during transfusion

Massive Transfusion

Adverse consequences of massive transfusion

ProblemMethods to Minimize
Hypothermia Cooled productsAppropriately re-warm products.
Use of blood warmers
Poor O2 delivery Depletion of 2,3 DPG in PRBC

Haemostatic abnormalities
Dilutional coagulopathyMonitor coagulation profile at appropriate intervals during massive transfusions.

Use of appropriate factors like FFPs, Cryoprecipitate during massive transfusions
Hypocalcaemia
Consumption with coagulopathy and bound to citrate added to transfused unitsMonitor and replace calcium as necessary
HypomagnesaemiaBound to citrate in transfused unitsMonitor and replace magnesium as necessary
Citrate toxicity Citrate is added to stored units as an anticoagulantMonitor for acidosis during massive transfusions
Lactic acidosisHyperlactataemia due to anaerobic metabolism in stored units


  • Storage medium pH ~5.5 for CPD

  • pCO2 in PRBC 150~200mmHg due to impermeable containers

  • New PRBC pH ~7.1

  • At 21 days pH ~6.9
  • Monitor for acidosis during massive transfusions
    HyperkalaemiaPotassium migrates from stored erythrocytes into plasma whilst in storage


  • Serum [K+] in PRBC ~ 90mmol/L

  • Mitigated by K+ loss during critical bleeding
  • Careful monitoring and usage of products within a specified period
    Air embolism Inadvertent infusionCareful monitoring, use of transfusion sets with air vents, filters

    JC / Sakurai 2019

    Exam appearances

    ExamExact wordingRelationshipSuccess
    2020A Q03 Outline the potential adverse consequences of blood transfusion. historical_member
    2013B Q13 Outline the adverse consequences of a blood transfusion. (75% of marks) Define massive blood transfusion and list the adverse consequences associated with a massive blood transfusion. (25% of marks) historical_member
    2017A Q16 List the potential problems resulting from blood transfusion and methods used to minimise them. historical_member
    2025B Q06 Outline the potential adverse consequences of blood product transfusion including the underlying mechanisms. variant 16.00%
    2026A Q06 Outline the potential adverse effects of red blood cell transfusion. Effects secondary to massive transfusion are NOT required. variant 53.00%