Canonical Question

Anti-anginal

V5 D7.iii Historical V4 G7.iv 1 appearance

Master answer

Myocardial ischaemia

METOPROLOLGLYCERYL TRINITRATE
β1 selective adrenoceptor antagonistNitrate
MoA β1 adrenoceptor = GsPCR
In Myocardium

–        Inhibition → Decreased activation of adenylyl cyclase → Decreased intracellular [cAMP] → Decreased activation of protein kinase A → Decreased phosphorylation and activation of intracellular enzymes → Decreased intracellular [Ca2+]

–        Decreased inotropy and decreased chronotropy → Decreased myocardial oxygen consumption → Relief of ischaemia
Nitrate groups activated by thiols to nitric oxide

In vascular smooth muscle (veins > arteries)

–        → Diffuses intracellularly and activated guanylyl cyclase → increased intracellular [cAMP] → Decreased intracellular [Ca2+] → decreased vascular tone → venodilation and vasodilation

–        → Decreased preload and decreased afterload

–        → Decreased myocardial O2 consumption → Relief of ischaemia
A/ECVS
–        Bradycardia
–        Hypotension
–        Arrhythmia
–        Decompensation of acute heart failure → Cardiogenic shock
–        Rebound myocardial ischaemia on abrupt cessation
 
Resp
–        Bronchospasm if reactive airways disease
 

CNS
–        Vivid dreams
–        Lethargy
 
Other
–        Dyslipidaemia
–        Hyperglycaemia
CVS
–        Hypotension
–        Reflex tachycardia
–        If preload dependent cardiac output → decreased cardiac output → shock
 


Resp
–        Methaemoglobin → decreased O2 delivery
–        Bronchodilation → V/Q mismatch
 
CNS
–        Increased cerebral blood flow
 
Other
–        Tolerance

Sakurai 2016

Exam appearances

ExamExact wordingRelationshipSuccess
2010A Q16 Define the mechanisms of action and adverse effects of metoprolol and glyceryl trinitrate when used to manage myocardial ischaemia. historical_member